All of the evidence provided on Hippocampome.org are quotes from scientific texts.
However, because quoted passages may be difficult to understand in isolation, contextual information and expanded abbreviations set in square brackets have been added for clarity.
"The Neurobiotin-labeled axon was immunonegative for PV [parvalbumin], CR [calretinin], CCK [cholecystokinin], VIP [vasoactive intestinal polypeptide], vAChT [vesicular acetylcholine transporter], ChAT [choline acetyltransferase], GlyT2 [glycine transporter 2], CB1 [cannabinoid receptor 1], and GAD [glutamic acid decarboxylase] (tested with two antibodies). The latter is surprising, but judging from the relatively low level of GAD [glutamate decarboxylase] mRNA in the soma of many ENK[enkephalin]positive cells, the terminals may express levels of GAD undetectable with our methods and antibodies. The axon terminals tested
for VIAAT [vesicular inhibitory amino acid
transporter] were immunopositive (Fig. 2B)."
EXPRESSION: negative (inference)
ANIMAL: mouse
PROTOCOL: immunohistochemistry
Page location: p49, Table 2
"Table 2. Co-localization between neuropeptides and calcium-binding proteins"
Interpretation notes: Glutamatergic and GABAergic types in CA1 SLM_SR are negative for SOM
EXPRESSION: negative (inference)
ANIMAL: rat
PROTOCOL: immunohistochemistry
Page location: p392, right bottom
"Hippocampus. SOM [somatostatin]-immunoreactive cell bodies are primarily
confined to stratum oriens in the CA1 region, with only a small number of cells in stratum pyramidale, and practically none in
strata radiatum and lacunosum-moleculare (Fig. 20). They are
more widespread in the CA3 region and frequently occur in strata
pyramidale, lucidum, and proximal radiatum."
Interpretation notes: Glutamatergic and GABAergic types in CA1 SLM_SR are negative for SOM
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positive-negative: unresolved inferential conflict
